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肾脏在很大程度上参与了充血性心衰的发病机理。心衰时,心脏排血机能障碍引起机体自稳性机制发生复杂的变化,在调整过程中调节细胞外液渗透压和容量的血流动力学和激素机制改变,导致肾脏排泄盐和水的能力降低。氯化钠与水潴留,细胞外液容量增加,随之发生循环充血和水肿。本文对心脏病人水肿形成的一些生理性变化进行综述。心脏丧失唧筒功能时,它在正常充盈压下的排血量,不能满足外周组织对氧与其他营养的需要。
The kidney is largely involved in the pathogenesis of congestive heart failure. Heart failure, cardiac discharge dysfunction causes complex changes in the body’s self-stability mechanism, adjusting the process of regulation of extracellular fluid osmotic pressure and volume changes in hemodynamics and hormone mechanisms, leading to renal excretion of salt and water capacity reduce. Sodium chloride and water retention, extracellular fluid volume increased, followed by congestion and edema. This article reviews some physiological changes in the edema of cardiac patients. When the heart loses the pump function, its amount of drainage under normal filling pressure can not meet the needs of peripheral tissues for oxygen and other nutrients.