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在冠状动脉粥样硬化性心脏病的发病机制学说中,除了已被人们接受的脂质浸润学说和损伤反应学说外,炎症反应的作用也逐渐被人们所认识。炎症反应贯穿于粥样硬化斑块的形成、演变以及破裂的整个病理过程。炎症细胞与血管内环境中的细胞、细胞因子、炎症介质等的相互作用极其复杂,
In the pathogenesis of coronary atherosclerotic heart disease theory, in addition to the accepted theory of lipid infiltration and damage response theory, the role of the inflammatory response is gradually being recognized. Inflammatory reactions throughout the formation of atherosclerotic plaques, evolution and rupture of the entire pathological process. The interaction of inflammatory cells with cells, cytokines, inflammatory mediators and the like in the intravascular environment is extremely complicated,