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目的探讨氧自由基(ROS)致Ca致突变作用,与胃Ca发生发展的关系。方法实验组:胃镜及病理确诊的胃Ca病例。对照组:胃镜及病理诊断为慢性浅表性胃炎病例,采用硫代巴比妥酸比色法检测胃粘膜组织中脂质过氧化反应终产物———丙二醛含量,从而间接推测机体内ROS水平。结果实验组LPO含量较对照组显著升高。结论氧自由基与胃Ca发生发展确有密切关系,有致Ca致突变作用。
Objective To explore the role of Ca (OH) 2 induced by oxygen free radicals (ROS) in the development of gastric Ca. Methods Experimental group: gastroscopy and pathological diagnosis of gastric Ca cases. Control group: gastroscopy and pathological diagnosis of chronic superficial gastritis cases, the use of thiobarbituric acid colorimetric assay of gastric ulcer lipid peroxidation product --- malondialdehyde content, which indirectly speculated that the body ROS level. Results The content of LPO in experimental group was significantly higher than that in control group. Conclusion Oxygen free radicals are closely related to the occurrence and development of gastric Ca, causing Ca-induced mutagenesis.