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［目的］探讨嗜铬细胞瘤合并肾动脉狭窄的发病机制、肾血管造影的表现，与儿茶酚胺、肾素的关系及对肾血管的作用。［方法］分析个案报告和复习文献。［结果］患者为左肾上腺嗜铬细胞瘤合并双侧肾动脉狭窄，恶性高血压，２４小时尿儿茶酚胺升高，切除肿瘤后血压下降但未降至正常。［结论］本病的发病机制可能为肿瘤压迫或纤维粘连合并儿茶酚胺过度分泌引起的肾动脉可逆或不可逆性狭窄。由于引起肾动脉狭窄的病因不同，血管造影可有不同的表现。与儿茶酚胺的过度分泌及肾动脉狭窄对高血压有不同的致病作用。
[Objective] To investigate the pathogenesis of renal stenosis with pheochromocytoma and the manifestation of renal angiography, and its relationship with catecholamines, renin and effects on renal vasculature. [Methods] Analyze case reports and review literature. [Results] The patient had left adrenal pheochromocytoma combined with bilateral renal artery stenosis, malignant hypertension, and elevated 24-hour urinary catecholamines. After resection of the tumor, the blood pressure decreased but did not return to normal. [Conclusion] The pathogenesis of this disease may be reversible or irreversible stenosis of the renal artery caused by tumor compression or fibrosis with excessive secretion of catecholamines. Due to the different causes of renal artery stenosis, angiography can have different manifestations. Excessive secretion of catecholamines and renal artery stenosis have different pathogenic effects on hypertension.