行为性应激和/或增加饮食钠摄入可能参与原发性高血压病的发病。与此假说有关的实验室研究已经显示行为性应激或增加钠摄入可使具有遗传性易感的动物出现或加重实验性高血压。然而,让遗传上血压正常的动物长期接受行为性应激或高钠负荷的影响,欲使其出现高血压的努力却产生了可疑的结果。因此,对这些环境—行为变量在长期血压控制中的作用的 Behavioral stress and / or increased dietary sodium intake may be involved in the pathogenesis of essential hypertension. Laboratory studies related to this hypothesis have shown that behavioral stress or increased sodium intake can cause or exacerbate experimental hypertension in genetically predisposed animals. However, trying to make a person with a genetically normal blood pressure long-term exposure to behavioral stress or high sodium load, trying to make it hypertensive, has yielded suspicious results. Therefore, the role of these environment-behavioral variables in long-term blood pressure control