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血管平滑肌细胞(VSMC)的过度增殖和迁移是多种心血管疾病共同的病理基础。VSMC不仅具有调节血管收缩的作用,同时还参与细胞外基质的合成与分泌,进而影响血管功能[1-2]。
Over-proliferation and migration of vascular smooth muscle cells (VSMCs) are a common pathological basis for a variety of cardiovascular diseases. VSMC not only has the role of regulating vasoconstriction, but also involved in the synthesis and secretion of extracellular matrix, thus affecting vascular function [1-2].