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已知吸烟、血脂升高和高血压是增加主动脉和冠状动脉的粥样硬化斑块的原因,但这些危险因子对各个斑块的结构和成份的影响并不够了解。触发冠心病人发生急性心脏事件的常见因子是血栓。覆盖斑块的内膜剥露(糜烂)或斑块撕断(破裂),使斑块中心(致血栓性高)与血管腔的血液接触而发生血栓。这两种过程是不同的,内皮剥露后,血栓形成于斑块表面,而斑块破裂后血栓的成分中还有破裂斑块的脂质中
It is known that smoking, hyperlipidemia, and hypertension are responsible for increased atherosclerotic plaques in the aorta and coronary arteries, but the effects of these risk factors on the structure and composition of each plaque are not well understood. A common factor triggering acute cardiac events in patients with coronary heart disease is thrombosis. Cover the plaque intimal debridement (erosion) or plaque tear (rupture), so that the center of the plaque (caused by high thrombus) and vascular blood vessels in contact with the occurrence of thrombosis. These two processes are different, after endothelial disruption, thrombosis is formed on the surface of the plaque, and after plaque rupture there is also a ruptured plaque lipid in the composition of the thrombus