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本文依据急性下壁和/或后壁心梗伴胸导 ST 段的变化,将病例分为两组。A 组27例胸导联 ST 段无明显改变;B 组15例为2个胸导联以上 ST 抬高≥0.2mV。结果显示:B组较 A 组 QRS 记分及 GOT 峰值明显增高(P<0.01);住院期间 B 组较 A 组严重合并症、梗塞后心绞痛、再梗塞及死亡率均增高。本文提示伴对应胸导联 ST 段抬高的急性下壁和/或后壁心梗属高危亚组。其发生机制可能与多支血管病变及病变血管通过侧支循环发生“盗血”现象有关。
This article based on acute inferior wall and / or posterior wall myocardial infarction with ST-segment changes in the chest, the cases were divided into two groups. A group of 27 cases of chest lead ST segment no significant change; B group of 15 cases of 2 chest lead above ST elevation ≥ 0.2mV. The results showed that the score of QRS and the peak of GOT in group B were significantly higher than those in group A (P <0.01). Serum complication, post-infarction angina, re-infarction and mortality in group B were significantly higher than those in group A during hospitalization. This article suggests that the acute inferior wall and / or posterior wall myocardial infarction with ST segment elevation corresponding to the chest lead is a high-risk subgroup. Its mechanism may be related to multi-vessel disease and vascular lesions through the collateral circulation “steal” phenomenon.