试验研究已经显示急性脑损伤可引起系统抗炎反应, 包括单核细胞失活和细胞免疫反应降低 [1]。血液中抗炎细胞活素的高表达在脑介导的外周免疫抑制方面起重要作用。本研究设脑出血组和对照组, 检测脑出血引起的来自单核细胞 (白介素-6和白介素-10) 或来自T细胞 (白介素-13) Experimental studies have shown that acute brain injury can cause systemic anti-inflammatory reactions, including monocyte inactivation and decreased cellular immune response [1]. The high expression of anti-inflammatory cytokines in the blood plays an important role in brain mediated peripheral immunosuppression. In this study, ICH and control groups were treated with monocytes (interleukin-6 and interleukin-10) or T-cells (interleukin-13)