心肌梗塞时心交感神经兴奋,儿茶酚胺大量释放,加重缺血心肌损伤并诱发心律失常。相反,利血平耗竭儿茶酚胺后,则可减轻心律失常。近年研究指出,缺血心肌内肾上腺素α、β受体密度增加,α、β受体阻断剂均有抗缺血心律失常作用。亦有哌唑嗪、普萘洛尔对缺血心律失常无效的不同报道。本实验目的旨在进一步证实并比较肾 Heart sympathetic excitement during myocardial infarction, catecholamine release a large number of increased myocardial ischemia and induced arrhythmia. In contrast, after reserpine depleted catecholamines, can reduce arrhythmia. Recent studies have pointed out that ischemic myocardial adrenaline α, β receptor density increased, α, β blockers have anti-ischemic arrhythmia. There are also prazosin, propranolol different reports of ischemic arrhythmia invalid. The purpose of this experiment is to further confirm and compare the kidneys