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Campbell等描述了一种主要是由于变性的红细胞—血影细胞堵塞滤帘孔道而引起的青光眼,其发病与陈旧性玻璃体出血有关。手术或外伤破坏了玻璃体前膜,此变性血细胞进入前房而均可发病。其临床特点为:疼痛、眼压升高、角膜水肿、前房内有大量棕色细胞、无Kp、有时在房角及滤帘上有一薄层棕色色素。术后数天或数周眼压可升高到40—70mmHg,并持续数周至数月。可用抗青光眼药物、前房或玻璃体冲洗或睫状体冷冻治疗。作者报告20例因持续一年的玻璃体出血施行封闭式玻璃体切除术,术后观察6个月,眼压超过30mmHg者定为术后青光眼。
Campbell et al. Describe a type of glaucoma mainly caused by the blockage of the pores of the filter cell by a denatured erythrocyte-ghost cell whose onset is associated with old vitreous hemorrhage. Surgery or trauma destroyed the anterior vitreous membrane, the degeneration of blood cells into the anterior chamber and can be disease. Its clinical features are: pain, elevated intraocular pressure, corneal edema, anterior chamber with a large number of brown cells, no Kp, and sometimes in the corner and the curtain has a thin layer of brown pigment. Intraocular pressure can be increased to 40-70 mmHg for days or weeks after surgery, and continues for weeks to months. Available anti-glaucoma drugs, anterior chamber or vitreous flush or ciliary body cryotherapy. The authors report 20 cases of closed vitrectomy with vitreous hemorrhage for one year. Postoperative observation of 6 months, intraocular pressure more than 30mmHg were identified as postoperative glaucoma.