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以‘富士’、‘国光’苹果的花粉为试材，研究肌醇磷脂信息传递系统在苹果花粉萌发和花粉管生长中的作用。结果显示，肌醇磷酸脂酶抑制剂Ｌｉ＋ 、蛋白质激酶抑制剂Ｈ７ 能显著抑制花粉萌发和花粉管生长，且随其浓度提高，抑制效应增强；Ｌｉ＋ 的效应可被外加肌醇逆转。磷脂酶（ＰＬＣ）抑制剂新霉素仅在高浓度时显著抑制花粉萌发和花粉管生长。此外，上述抑制剂在高浓度时对花粉萌发的初始阶段具显著的抑制效果。加速磷脂酰肌醇分解的脱氧胆酸钠能显著抑制花粉萌发和花粉管生长，而促进肌醇磷脂降解的２ ，４Ｄ的作用很小。由此表明，肌醇磷脂（ＰＩ） 信息传递系统可能参与了苹果花粉萌发和花粉管生长过程，并可能在花粉萌发的启动过程中具重要调节作用。
The effects of inositol signaling system on the pollen germination and pollen tube growth of apple were studied using the pollen of ’Fuji’ and ’Guoguang’ apples. The results showed that inositol phosphatase inhibitor Li + and protein kinase inhibitor H7 significantly inhibited pollen germination and pollen tube growth. With the increase of its concentration, the inhibitory effect was enhanced. The effect of Li + could be reversed by extraneous inositol. Neomycin, a phospholipase (PLC) inhibitor, significantly inhibited pollen germination and pollen tube growth only at high concentrations. In addition, the above-mentioned inhibitors have a significant inhibitory effect on the initial stage of pollen germination at high concentrations. Accelerated phosphatidylinositol decomposition of sodium deoxycholate can significantly inhibit the pollen germination and pollen tube growth, and promote the degradation of inositol phospholipid 2, 4 D little effect. Thus, inositol phospholipid (PI) signaling system may be involved in the process of pollen germination and pollen tube growth in apple and may play an important regulatory role in the initiation of pollen germination.