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肾脏的抗高血压作用肾脏加压系统或肾-肾上腺系统(肾素-血管紧张素-醛固酮系统)是实验性或人类原发性高血压持续血压升高的重要原因。但有些实验性高血压,人的肾血管性高血压,特别是大多数原发性高血压者,血浆肾素和血管紧张素或醛固酮并不升高;此外,使动物对血管紧张素Ⅱ产生高度免疫后,并不能防止与纠正其肾血管性高血压;用多肽抑制剂以阻断血管紧张素的作用后,只能使对侧肾完整的实验
Antihypertensive effects of the kidneys Renal pressure system or the adrenal system (renin-angiotensin-aldosterone system) is a major cause of persistent elevated blood pressure in experimental or human essential hypertension. However, some experimental hypertension, human renal vascular hypertension, especially in most patients with essential hypertension, plasma renin and angiotensin or aldosterone does not increase; in addition, the animals on angiotensin II production After a high degree of immunity, does not prevent and correct its renovascular hypertension; with peptide inhibitors to block the role of angiotensin, can only make contralateral renal integrity of the experiment