论文部分内容阅读
七、原发性醛固酮增多症并发高血压低血钾危象(一)病因原发性醛固酮增多症(简称原醛),大多(90%)由于肾上腺皮质腺瘤、少数由于皮质增生或癌肿所引起。由于自主性分泌醛固酮等盐皮质激素过多,潴钠排钾,使血压升高和血钾减低,可出现高血压低血钾瘫痪危象。(二)临床表现原醛的特征是高血压伴有低血钾,以及由于低血钾所引起的肌肉软弱麻痹和失钾性肾病。1.高血压,是最早出现的主要症状,常阵发性加重,多在150~250/90~130mmHg。常伴
Seven, primary aldosteronism complicated by high blood pressure and hypokalemia crisis (a) etiology of primary aldosteronism (referred to as primary aldosterone), mostly (90%) due to adrenocortical adenoma, a few due to cortical hyperplasia or cancer Caused. Due to the autonomous secretion of aldosterone and other mineralocorticoid excess potassium retention potassium row, so that blood pressure and serum potassium decreased, there may be high blood pressure hypokalemia paralysis crisis. (B) the clinical manifestations of the original aldehyde is characterized by hypertension associated with hypokalemia, as well as due to hypokalemia caused by muscle weakness paralysis and K of nephropathy. 1. Hypertension, is the earliest major symptoms, often paroxysmal aggravating, more in 150 ~ 250/90 ~ 130mmHg. Often accompanied