论文部分内容阅读
迄今,植物病毒病尚未研制出实用的化学治疗物或天然病毒抑制剂,因此进行植物病原侵染及寄主反应机理的研究,以探讨植物抗病性的本质,已日益引起重视。Loebenstein(1963)提出,植物对病毒的获得抗性机理,可以类似于动物中干扰素的诱导抗性。Ross(1961)接种TMV于枯斑三生烟(N.tabacum var.samsun NN)半叶或下部叶片,可诱导未接种半叶或上部叶片对病毒再次侵染的抗性;并推测这种系统获得抗性可能是由于接种部位产生一种或几种物质,系统转移到健康组织。此后,许多学者相继报道了几种与植物获得抗性有关的抗病毒物质,诸如:抗病毒因子(AVF),病理关联蛋白(PRs或b蛋白),病毒复制抑制剂(IVR),病毒抑制因子(VIR)等
So far, no practical chemotherapeutic or natural virus inhibitors have been developed for plant virus diseases. Therefore, studies on plant pathogen infection and host reaction mechanism to explore the nature of plant disease resistance have drawn increasing attention. Loebenstein (1963) suggested that the mechanism by which plants acquire resistance to viruses may be similar to the induced resistance of interferons in animals. Inoculation of TMV on the half or lower leaves of N. tabacum var.samsun NN inoculated with Ross (1961) induced resistance to reinfection in the unvarying half or upper leaves, and speculated that such a system Acquired resistance may be due to inoculation sites produce one or several substances, the system transferred to healthy tissue. Since then, many scholars have successively reported several antiviral substances related to plant acquired resistance, such as antiviral factor (AVF), pathological related protein (PRs or b protein), viral replication inhibitor (IVR), virus inhibitory factor (VIR) and so on