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在大多数临床学家的文章中,氟中毒只联想到骨组织,这一事实在较广泛地用它于中毒分级中反映出来。但这种概念既同氟的生物和毒理性质相矛盾,又同慢性氟中毒的发病机理相矛盾。作为生物微量元素,氟类的其它微量元素,它不能限制其作用于某一系统的器官,它的影响很广泛。生理浓度的氟促进磷酸钙盐的形成,对许多重要的酶的活性发生影响,从而影响到组织代射过程的速度和方向,以维持内环境的稳定。过量的氟进入机体导致中毒发生的危险。吸入氟化物的毒性较经口进入的毒性
In most clinical scientists’ articles, fluorosis is associated only with bone tissue, a fact that is more widely reflected in its toxicological grade. However, this concept not only contradicts the biological and toxicological properties of fluorine, but also contradicts the pathogenesis of chronic fluorosis. As a biological trace element, fluorine other trace elements, it can not limit its role in a system of organs, it has a wide range of effects. Physiological concentrations of fluoride promote the formation of calcium phosphate salts, affecting the activity of many important enzymes, which affects the rate and direction of tissue engraftment in order to maintain homeostasis. Excessive fluoride into the body leading to the risk of poisoning. Toxicity of fluoride inhalation than oral toxicity