梭曼0.9LD_(50)中毒的小鼠,经HI—6在不同时间治疗(立即,5分,10分,20分,30分),对被梭曼抑制的全血胆碱酯酶均有一定的重活化作用。中毒对照组胆碱酯酶活力为正常对照组活力的18.75%,立即治疗及5分,10分,20分,30分治疗组的胆碱酯酶活力分别为51.50%,36.78%,31.89%,26.27%及25.92%。所以,HI—6延迟治疗对提高胆碱酯酶活力是有效的。 Soman 0.9 LD50 poisoned mice were treated with HI-6 at different times (immediately, 5 minutes, 10 minutes, 20 minutes, 30 minutes), and all the blood cholinesterase inhibited by soman Certain reactivation effect. The cholinesterase activity of the poisoning control group was 18.75% of that of the normal control group. The cholinesterase activities of the treatment groups immediately treated with 5, 10, 20 and 30 minutes were 51.50%, 36.78% and 31.89%, respectively. 26.27% and 25.92% respectively. Therefore, delayed treatment of HI-6 is effective in increasing cholinesterase activity.