PMN受到刺激后可表现为凋亡、原发性坏死、继发性坏死、凋亡延迟 (存活 ) ,而创伤后细胞凋亡的发生在机体内是一种普遍现象。PMN凋亡延迟与过度炎症反应及组织器官损伤有密切关系 ,调节PMN凋亡可以促进炎症的吸收和控制。凋亡机制的实现不仅需要PMN自身参与 ,也需要巨噬细胞或组织细胞对凋亡细胞识别和处理 After being stimulated, PMN can be expressed as apoptosis, primary necrosis, secondary necrosis, delayed apoptosis (survival), and the occurrence of post-traumatic apoptosis in the body is a common phenomenon. PMN apoptosis is closely related to excessive inflammatory reaction and tissue and organ damage. Regulation of PMN apoptosis can promote the absorption and control of inflammation. The realization of apoptosis mechanism needs not only the participation of PMN itself, but also the identification and treatment of apoptotic cells by macrophages or tissue cells