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在感染了病原物的植物组織的代謝研究中曾注意到呼吸強度和葡萄糖异化途径的改变。現在已明确地肯定了在受感染的寄主組織中,特別是在銹菌感染的情况下(Allen1959;Daly和Sayre 1957;Samborski和Shaw 1956;Farkas和Kiraly 1955)呼吸強度是增加的。感染病毒的植物在这方面的一些結果則还未能肯定(Bawden 1959;Millerd和Scott 1962)。更有兴趣的是在锈菌感染的研究中发現葡萄糖异化途径由酵解(EMP)轉向戊糖磷酸(HMP)途径(Daly等1962;Shaw和Samborski 1958),根据回收的CO_2中同位素碳的比例(Bloom和Stetten 1953)来确定的这一現象还未在真菌以外的其他寄生物——
Metabolic studies of plant tissues infected with pathogens have noticed changes in respiratory intensity and glucose-alienation pathways. It has now been unequivocally affirmed that respiration intensity is increased in infected host tissues, especially in the case of rust infection (Allen 1959; Daly and Sayre 1957; Samborski and Shaw 1956; Farkas and Kiraly 1955). Some of the results of this virus-infected plant in this area have yet to be confirmed (Bawden 1959; Millerd and Scott 1962). Of greater interest is the finding that the pathways of the glucose-alienation shift from glycolysis (EMP) to the pentose phosphate (HMP) pathway in studies of rust infection (Daly et al. 1962; Shaw and Samborski 1958). Based on the recovery of isotopic carbon in CO2 The proportion (Bloom and Stetten 1953) to determine this phenomenon has not yet in addition to fungi other parasites -