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慢性肺源性心脏病(简称肺心病),在肺心功能失代偿期时,常伴有不同程度的肾功能损害,尤其是急发期。利用血、尿、β_2—微球蛋白(β_2—MG)测定来观察肺心病肾功能改变,特别是肾小管功能,优于血肌酐(Cr)和尿素氮(BUN)的检测。本文通过40例对比研究结果,所有肺心病急发期患者,血、尿、β_2—MG 均增高,缓解期血β_2—MG 均大于正常,而尿β_2—MG 有7例(占17.5%)恢复正常。BUN 在急发期>20mg/dl 者25例(占62.5%)到缓解期降至5例(占12.5%),Cr 在急发期>1.5mg/dl 者13例(占32.5%),缓解期下降到4例(占10%)。对肺心病致肾功能不全的判断,我们认为以血、尿β_2—MG最为敏感可信。
Chronic pulmonary heart disease (referred to as pulmonary heart disease), pulmonary heart failure, often accompanied by varying degrees of renal dysfunction, especially in emergency. The use of blood, urine and β_2-microglobulin (β_2-MG) measurement to observe the changes of renal function in patients with pulmonary heart disease, especially the renal tubular function, better than the serum creatinine (Cr) and urea nitrogen (BUN) detection. In this paper, 40 cases of comparative study results, all patients with acute pulmonary heart disease, blood, urine, β 2-MG were elevated, β 2-MG were significantly higher than normal, and urinary β 2-MG in 7 cases (17.5%) recovery normal. BUN was reduced from 25 cases (62.5%) to remission stage to 5 cases (12.5%) in the acute phase of> 20mg / dl, and 13 cases (32.5%) of Cr in the acute stage> 1.5mg / dl Period down to 4 cases (10%). Pulmonary heart disease caused by renal insufficiency, we believe that the blood, urine, most sensitive to β_2-MG credible.