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左旋精氨酸(L-Arginine,L-Arg)是合成一氧化氮(NO)的底物,它可刺激血管内皮细胞的NO合成酶(NOS)而增加NO的合成和释放[1]。随着人们对NO的了解逐步深入,L-Arg在临床和基础的研究和应用更加广泛。本文对近年来L-Arg的心肌保护机制和心血管疾病中的研究进展作一综述。1L-Arg的保护作用L-Arg的保护作用是通过产生和释
L-arginine (L-Arginine), a substrate for the synthesis of nitric oxide (NO), stimulates NO synthase (NOS) in vascular endothelial cells and increases NO production and release [1]. With the gradual deepening of the understanding of NO, L-Arg is more widely used in clinical and basic research. This article reviews the research progress of L-Arg myocardial protection mechanism and cardiovascular disease in recent years. Protection of 1L-Arg The protective effect of L-Arg is through the generation and release