急性心肌梗死病人早期即可出现心室重构,心功能下降或呈下降趋势。作为代偿机制,神经-内分泌-免疫系统及内皮系统被迅速激活,在短期内有利于维持机体的血流动力学稳定。但持续性过度应激往往会造成一系列病理后果,进而损害心功能,导致心力衰竭的发生。 Acute myocardial infarction in patients with early ventricular remodeling may occur, decreased or decreased cardiac function. As a compensatory mechanism, the neuroendocrine-immune system and the endothelium system are rapidly activated, which helps maintain the hemodynamic stability of the body in a short period of time. However, persistent over-stress often leads to a series of pathological consequences, thus impairing heart function and leading to heart failure.