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心力衰竭后期经常可见到有严重的水肿并伴有低钠血症。血液动力学变化在此种水肿发生上的意义尚未清楚;抗利尿素在此过程中的作用也未阐明。曾有学者提出抗利尿素(ADH)可能参予充血性心力衰竭“稀释性低渗症候群”的发病机制。为了查明此种可能性。本文研究了20名重症充血性心力衰竭病人的血浆ADH浓度及其与血液动力学、血浆渗透压的关系。结果表明一组病人有不适当的高ADH血症(14.5±8.8pg/ml),其血浆渗透压显著地低于正
Severe edema and hyponatremia are often seen late in heart failure. The significance of hemodynamic changes in the occurrence of this edema is unclear; the role of antidiuretic in this process is not elucidated. Some scholars have proposed anti-diuretic (ADH) may participate in the pathogenesis of “dilutional hypotonic syndrome” in congestive heart failure. In order to find out this possibility. This article studied the plasma ADH concentrations in 20 patients with severe congestive heart failure and its relationship with hemodynamics and plasma osmotic pressure. The results showed that one group of patients had inappropriate hyper-ADH (14.5 ± 8.8 pg / ml), whose plasma osmolarity was significantly lower than positive