论文部分内容阅读
目的探讨0~3岁铁蛋白(SF)缺乏儿童上肢表面肌电的变化情况。方法选取2015年1月-2016年4月在南通市妇幼保健院儿童保健科门诊就诊的0~3岁幼儿240名为研究对象,其中SF正常儿童120名为对照组,SF缺乏儿童120名为试验组。采用双通道表面肌电信号采集系统,对240名幼儿的左右上肢13个基本动作主肌肉群的表面肌电信号进行采集,每个动作重复3次,分析两组儿童两侧上肢三角肌中部、肱二头肌、肱三头肌、掌长肌和指伸肌等肌肉的平均肌电和积分肌电的不同特性。结果对照组儿童肱二头肌、肱三头肌、掌长肌左右肢上表面肌电的平均振幅均高于试验组儿童,差异有统计学意义(P<0.05);对照组儿童三角肌中部的右上肢和肱二头肌、肱三头肌、掌长肌左右肢上表面肌电的积分肌电均高于试验组儿童,差异有统计学意义(P<0.05)。结论 SF缺乏儿童与SF正常儿童的上肢表面肌电存在明显差异。全社会均应积极关注幼儿铁缺乏的情况,为科学合理地对SF缺乏儿童进行早期、规范治疗提供依据。
Objective To investigate the changes of myoelectricity in the upper extremities of children with 0-3 year old ferritin (SF) deficiency. Methods From January 2015 to April 2016, 240 children aged 0 ~ 3 years old from outpatient department of Child Health Care Hospital of Nantong Maternal and Child Health Care Hospital were enrolled. Among them, 120 children with normal SF were control group and 120 children with SF deficiency were test group. Using the dual-channel surface EMG signal acquisition system, the surface EMG signals of the 13 basic movements of the left and right upper limbs of 240 young children were collected. Each action was repeated 3 times. The middle part of the upper limb deltoid muscle of both groups was analyzed. Biceps, triceps, palmar longus, and extensor muscles and other muscles of the average EMG and integral myoelectric different characteristics. Results The average amplitude of EMG in the left and right limbs of the biceps, triceps and palmar longissimus muscles of the control group was significantly higher than that of the test group (P <0.05). The mean central part of the deltoid muscle The right upper limbs and biceps, triceps, palmar longus muscles on the left and right upper limb myoelectric integral electromyography were higher than the test group children, the difference was statistically significant (P <0.05). Conclusion There is a significant difference in the myoelectricity of the upper extremities of SF-deficient children and SF-normal children. All sectors of society should pay close attention to the situation of iron deficiency in children and provide the basis for scientifically and reasonably early and standardized treatment of SF-deficient children.