论文部分内容阅读
环斑花叶病侵染的番木瓜叶片暗呼吸速率为1.85μmol·O_2·dm~(-2)·min~(-1),较健叶增高63.7％,但光下依赖CO_2的氧释放速率较健叶低33.6％。表明环斑花叶病侵染叶片引起暗呼吸增高和抑制光下依赖CO_2的氧释放。在低光强(低于0.2m mol光量子·m~(-2)·S~(-1))病叶的氧释放速率(RO,μmol·O_2·dm~(-2)·min~(-1))为RO=-0.07+2.38PFD,而健叶的相应关系为RO=1.08+2.43PFO。表明氧释放速率与光强关系曲线的最初直线斜率,健叶的较病叶的高;氧释放速率随光强的增高而上升得较快。环斑花叶病侵染亦引起叶绿体膜脂半乳糖甘油脂和酰脂相对含量降低。病原的浸染可能引起叶绿体膜脂部分降解。
The dark respiration rate of papaya leaves infected by rodent mosaic virus was 1.85μmol · O 2 · dm -2 · min -1, which was 63.7% higher than that of healthy leaves, but the oxygen release rate of CO 2 dependent on light 33.6% lower than the healthy leaves. The results showed that the infected leaf lobed leaves caused dark breathing and inhibited the light-dependent CO 2 release. The oxygen release rate (RO, μmol · O 2 · dm -2 · min ~ (-1)) in diseased leaves of low light intensity (less than 0.2 m mol photon · m -2 · S -1) 1)) is RO = -0.07 + 2.38PFD, while the corresponding relationship of healthy leaves is RO = 1.08 + 2.43PFO. The initial linear slope of the oxygen release rate vs. light intensity curve is shown to be higher for the healthy leaf than for the diseased leaf; the rate of oxygen release rises faster with increasing light intensity. Ringspot mosaic infection also caused chloroplast membrane lipid galactosyl and lipid relative content decreased. Pathogen contamination may cause partial degradation of chloroplast membrane lipids.