论文部分内容阅读
“铁春1号”小麦耐小麦白粉病的机制是:①光合补偿作用:“铁春1号”小麦旗叶的表现光合速率下降较小,而真正光合速率比健康植株高,表明侵染点间的绿色组织具有光合补偿作用,一定程度上弥补病原菌的破坏与消耗。②超氧化物歧化酶活性增强,能及时清除因病害胁迫产生的超氧自由基(O_2·),避免了O_2对细胞膜的伤害。“铁春1号”膜磷脂种类在染病后没有变化,而感病品种膜磷脂在染病后增加了2个组份。③贮存物质运转的补偿作用:“铁春1号”在病害胁迫下,叶、茎、叶鞘中的干物质,以及氮、磷等养分的运出率增大,通过充分利用贮存物质部分的补偿光合产物低之不足。最终使籽粒中氮、磷及碳水化合物不减少。
The mechanism of wheat resistance to wheat powdery mildew was the following: (1) Photosynthetic compensation: the photosynthetic rate of flag leaf of “Tiantang 1” decreased less and the true photosynthetic rate was higher than that of healthy plants, indicating that the infection point Between the green tissue photosynthesis compensation role, to some extent, to make up for the destruction and consumption of pathogens. ② The activity of superoxide dismutase is enhanced, the superoxide radical (O 2 ·) produced by disease stress can be eliminated in time, and the injury of O 2 to cell membrane can be avoided. “Tieshuang No. 1” membrane phospholipids species did not change after the disease, and susceptible varieties of membrane phospholipids after the disease increased by 2 components. ③ Compensation function of storage material operation: Under the stress of “Tiantang 1”, dry matter in leaves, stems and leaf sheaths, as well as the outflow rate of nutrients such as nitrogen and phosphorus increased. By making full use of the compensation of the storage material part, Photosynthetic products low enough. Eventually make the grain of nitrogen, phosphorus and carbohydrate does not reduce.