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以质膜 Ca2 +通道阻断剂 L a Cl3和钙调素 (Ca M)拮抗剂氯丙嗪 (CPZ)对水稻幼苗预处理以阻碍 Ca2 +· Ca M信使传导后 ,研究了盐胁迫下 ,稻苗保护酶活性的变化。结果表明 :0 .15 mol/ L Na Cl胁迫下 ,L a Cl3和 CPZ预处理显著加剧稻苗过氧化氢酶 (CAT)活性和抗坏血酸过氧化物酶 (APX)活性下降 ;对谷胱甘肽还原酶 (GR)活性则无明显影响 ;而超氧化物岐化酶 (SOD)和过氧化物酶 (POD)活性前期无明显变化 ,仅在胁迫后期明显下降。盐胁迫下 ,L a Cl3和 CPZ预处理加剧 SOD、 CAT和 APX活性下降与加剧稻苗 MDA含量积累呈显著或极显著正相关。这暗示 Ca2 + · Ca M信使系统可能通过调节一些抗氧化酶的活性来调节稻苗对盐逆境的适应性
After pretreatment of rice seedlings with Ca (superscript 2+) channel blockers L a Cl3 and CP2, which block the messenger of Ca2 + · Ca M, Changes of protective enzyme activity in rice seedlings. The results showed that: L NaCl and CPZ pretreatment significantly inhibited the activity of catalase (CAT) and the activity of ascorbate peroxidase (APX) in rice seedlings under NaCl stress of 0.15 mol / L; the activity of glutathione However, the activities of superoxide dismutase (SOD) and peroxidase (POD) did not change significantly at the early stage and decreased only at the later stage of stress. Under salt stress, Lα Cl3 and CPZ pretreatment exacerbated the decline of SOD, CAT and APX activities and aggravate the accumulation of MDA content of rice seedlings showed a significant or very significant positive correlation. This suggests that the Ca2 + · Ca M messenger system may regulate the adaptability of rice seedlings to salt stress by regulating the activity of some antioxidant enzymes