血小板和凝血系统的激活是止血的生理过程,但也可变成病理过程。因此,更好地阐明血小板激活的机理及其与凝血系统的相互作用,探索药物如何才能阻止这些作用是有重要意义的。正常和受刺激内皮细胞层的抗血栓与致血栓作用正常内皮细胞层无致血栓作用,可能是其主动功能。内皮细胞所具有的负电荷能排斥血小板,所产生的前列腺环素(PGI_2)较血管壁的其它细胞为多,其浆膜中的血栓调节素为 Platelet and coagulation system activation is a hemostatic physiological process, but also can become a pathological process. Therefore, to better elucidate the mechanism of platelet activation and its interaction with the coagulation system, it is important to explore how drugs can prevent these effects. Normal and stimulated endothelial cell layer of anti-thrombotic and thrombotic effect of normal endothelial cell layer-induced thrombosis, may be its active function. The negative charge of endothelial cells can exclude platelets, PGI 2 produced more than other cells in the vascular wall, the thrombomodulin in the serosa