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用四氯化碳、脂肪饲料、含乙醇饮料等复合因素复制的肝硬变门脉高压性瘀证大鼠，舌脉诊与人肝硬化所见相同。组织学变化有二；①舌腹面粘膜出现局灶性或节段性或广泛性空泡变性，严重者细胞凝固坏死、溶解、结构破坏；②随门－腔静脉侧支循环的建立，舌深静脉压升高。镜下见舌粘膜特别是腹面粘膜下毛细血管、微小静脉扩张瘀血，内皮细胞肥大增生，腔内红细胞聚集，舌肌间血管瘀血，偶见小动脉壁坏死，伴间质水肿。
With carbon tetrachloride, fat feed, alcohol-containing beverages and other complex factors of cirrhosis with cirrhosis of the portal vein stasis syndrome, tongue pulse diagnosis and liver cirrhosis seen the same. Histological changes in two; ① tongue or abdominal mucosa appeared focal or extensive vacuolar degeneration, severe cell coagulation necrosis, dissolution, structural damage; ② With the portal - vena cava collateral circulation was established, the tongue depth Venous pressure increased. Under the microscope, see the tongue mucosa, especially the ventral submucosal capillaries, venous dilatation and microcirculation, hypertrophy of endothelial cells, intraluminal erythrocyte aggregation, tongue muscle bleeding, occasionally arteriole necrosis, with interstitial edema.