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利用手术切断听神经的方法分离中枢与外周耳蜗的神经联系,观察分离前后 DPOAE 的变化。在本实验条件下,所有豚鼠均可引出 DPOAE,在同等刺激强度和 L_1=L_2的情况下,低频 f_0=1006Hz 的 DPOAE 强度大,而较高频 f_0=2011和 f_0=4 033Hz 的 DPOAE 强度较小。切断听神经对DPOAE 无影响,说明无噪声状态下中枢不参与、不影响 DPOAE 的产生和形成。打开豚鼠骨大泡,可引起 DPOAE 明显下降,经分析发现主要为本底噪声增加所致。低频刺激时噪声增加量多于高频刺激。噪声增加原因可能是失去了骨大泡对噪声的屏障作用,使噪声经鼓膜内侧面作用于外耳道内所致。
The method of surgery to cut off the auditory nerve was used to separate the neural connection between central and peripheral cochlea, and to observe the changes of DPOAE before and after separation. Under the experimental conditions, all of the guinea pigs can induce DPOAE. Under the same stimulus intensity and L_1 = L_2, the DPOAE intensity of low frequency f_0 = 1006Hz is large, while the DPOAE intensity of high frequency f_0 = 2011 and f_0 = 4033Hz is higher than that of DPOAE small. Cut off the auditory nerve on DPOAE no effect, indicating no noise central state involvement, does not affect the DPOAE production and formation. Open Guinea pig large bubble can cause DPOAE decreased significantly, the analysis found mainly due to increased background noise. Low-frequency stimulation noise increase more than high-frequency stimulation. The reason for the increase of noise may be the loss of the barrier effect of the bullae on the noise and the effect of the noise acting on the inner ear canal through the inner side of the tympanic membrane.