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一、发病机理 (一) 心肌收缩功能异常(左室收缩功能异常)。收缩功能减弱分为原发于心肌收缩无力及继发于压力或容量负荷过重两类。 (二) 心衰的外周因素 充血性心衰(CHF)时,有许多激素变化,如循环中儿茶酚胺增多,肾素、血管紧张素,醛固酮(RAA)及精氨酸血管加压素(ADP)增多。CHF的严重程度与循环中儿茶酚胺水平有关。RAA系统激活,在CHF发生、发展和治疗中均有重要意义。 (三) 化学机制
First, the pathogenesis (a) myocardial systolic dysfunction (left ventricular systolic dysfunction). Systolic dysfunction is divided into primary myocardial weakness and secondary to stress or volume overload two categories. (B) Peripheral Factors of Heart Failure There are many hormonal changes during congestive heart failure (CHF), such as increased circulating catecholamines, renin, angiotensin, aldosterone (RAA) and arginine vasopressin (ADP) increase. The severity of CHF correlates with circulating catecholamines. RAA system activation, in the occurrence of CHF, development and treatment are of great significance. (C) chemical mechanism