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It has been reported that in utero cigarette smoke exposure(CSE)induced detrimental effect on central chemoreception of neonatal rats.However,the underlying mechanism is unknown.We presume that in utero CSE may give rise to oxidative damages to pFRG,the key site for central chemoreception,leading to impairment of neonatal central chemoreception.The present study was carried out to testify our assumption above.