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The physiological control of feeding behavior and energy balance involves complicated modulating mechanisms in which different brain areas and transmitters/receptors play roles.Our recent research focuses on the intake inhibitory effects of gastrointestinal satiation signaling via endogenous hindbrain leptin receptor (LepR) and glucagon-like-peptide-1 receptor (GLP-1 R) activation (Zhao et al: Int J Obes 2012∶36∶1522-8.), and on the contribution of hindbrain Lepting receptor (LepRb) signaling to the control of food intake (Kanoski et al: Am J Physiol Endocrinol Metab; 303∶496-503).