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Background No-reflow phenomenon is a risk factor which severely compromises the benefits of coronary revascularization in patients with acute myocardial infarction.Inflammatory response, as an essential component of cardiac ischemia/reperfusion (I/R) injury, has been suggested to contribute to the myocardial no-reflow.Since nuclear factor kappa B (NF-kB) is a key mediator of inflammation, we reasoned that inhibition of NF-kB might reduce the extent ofno-reflow.