论文部分内容阅读
矽肺是以肺组织纤维化为主的全身性疾病,从已有实验性矽肺观察,矽结节的发生与发展大体分四个阶段;即肺巨噬细胞由于矽尘的刺激出现增生与吞噬形成细胞结节,继之是细胞的崩解与坏死,接着为纤维化形成,最后形成矽结节的透明样变。生物体是一个有机的整体。每阶段的病理变化过程在生化免疫上必然有所反应,根据其反应有很多国内外学者进行了单一多方面的研究。但是形成矽结节导致的生化免疫指标尚无单一项能特异地反应矽肺发生与发展情况已为从所周知。为了不失生化免疫指标在矽肺诊断、治疗、观察等诸多方面的意义,我们在以前学者的基础上,分别对来自本地区19例
Silicosis is a systemic disease characterized by pulmonary fibrosis. From the existing experimental silicosis observation, the occurrence and development of silicon nodules can be roughly divided into four stages: the proliferation and phagocytosis of lung macrophages due to the stimulation of silica dust Cell nodules, followed by cell disintegration and necrosis, followed by the formation of fibrosis, the final formation of transparent nodules of silicon change. The organism is an organic whole. The pathological changes of each stage inevitably respond to biochemical immunity, according to the response of many scholars at home and abroad conducted a single multi-faceted study. However, the formation of silicon nodules caused by biochemical indicators of immunity have not been a single specific reaction to the occurrence and development of silicosis has been well-known. In order to not lose the significance of biochemical immune index in the diagnosis, treatment, observation and many other aspects of silicosis, on the basis of previous scholars, we separately analyzed the clinical data of 19 cases