弥散性伤害抑制控制(DNIC)是中枢性疼痛调节系统的一部分,它依赖于脊髓和脊髓上机制。以前研究表明,纤维肌痛患者试验性疼痛时与正常对照组相比缺乏DNIC效应。DNIC对慢痛(second pain)较快痛有更大的作用。因此假设强的条件刺激可缩短慢痛的时间。 此试验旨在分别评估慢痛抑制的相关机制的作用,以这种方式引导受试者对实验或条件刺激的注意力。 Dispersive damage suppression control (DNIC) is part of the central pain regulation system, which relies on the spinal cord and spinal cord mechanisms. Previous studies have shown that patients with fibromyalgia experimental pain when compared with the normal control group lack of DNIC effect. DNIC has a greater effect on the pain of second pain. So suppose that strong conditioning can shorten the time of slow pain. The aim of this trial is to evaluate the effects of mechanisms related to chronic pain inhibition, respectively, in such a way as to guide the subject’s attention to experimental or conditioned stimuli.