论文部分内容阅读
新生儿缺氧缺血性脑病(HIE)是新生儿窒息、缺氧所致的严重并发症,重症可威胁新生儿生命,存活者易留下神经系统后遗症。近年研究表明,促红细胞生成素(EPO)具有神经保护、神经营养作用。本实验在新生大鼠缺氧缺血性脑损伤(HIBD)模型上观察外源性EPO对超氧化物歧化酶(SOD)、丙二醛
Neonatal hypoxic-ischemic encephalopathy (HIE) is a neonatal asphyxia, a serious complication caused by hypoxia, severe may threaten the life of newborns, survivors easily leave the nervous system sequelae. Recent studies have shown that erythropoietin (EPO) has neuroprotective and neurotrophic effects. In this study, we observed the effects of exogenous EPO on superoxide dismutase (SOD), malondialdehyde (MDA) in neonatal rats with hypoxic-ischemic brain damage (HIBD)