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变应性鼻炎所引起的鼻症状,部分是直接由鼻粘膜效应细胞的化学介质作用;部分为鼻效应细胞受乙酰胆硷的活化所引起。副交感神经末梢继感觉神经终端受化学介质的刺激后反射性地逸出了乙酰胆碱。Szentivanyi(1968)假设支气管哮喘病人的呼吸道过度敏感是由于下呼吸道植物神经系统不平衡,特别是由于不正常的β肾上腺素能受体。近年来,采用受体-结合测定,已能够直接测试植物神经受体的密度及亲和力。有报告从支气管哮喘病人的肺组
Nasal symptoms caused by allergic rhinitis, in part, are directly mediated by the chemical mediator of nasal mucosal effector cells; partly due to the activation of nasal effector cells by acetylcholine. Parasympathetic nerve terminals sensory nerve terminals following the stimulation of the chemical medium after the reflex escape of acetylcholine. Szentivanyi (1968) hypothesized that the airway hypersensitivity in patients with bronchial asthma is due to an imbalance in the lower respiratory tract autonomic nervous system, especially due to abnormal beta adrenergic receptors. In recent years, receptor-binding assays have made it possible to directly test the density and affinity of autonomic nerve receptors. There are reports from the lungs of patients with bronchial asthma