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有许多证据表明,癌瘤的形成是一“多步骤”的变化过程.当细胞遭到一系列侵袭时,例如,受病毒感染后又有化学方面的攻击,癌瘤就可发生。可疑的是:这种影响破坏了细胞控制某些基因的能力,即所谓癌基因。因此,“多步骤”理论预示,2个或2个以上被激活的癌基因可能经常产生协同作用而致癌。 1985年10月中,在巴黎召开的协同基因作用讨论会上报道了巴斯德研究所DominiqueStehelin的研究,表明了两个癌基因之间的上述相互作用.癌基因之间的协同作用曾于1983年首次报道,只有当ras和myc癌基因共同作用时,才能转化成纤维细胞(Nature304卷,
There is a lot of evidence that the formation of cancerous tumors is a “multi-step” process of change. When a cell is attacked by a series of attacks, for example, after being infected by a virus, there is a chemical attack and cancer can occur. Suspectedly: this effect destroys the ability of cells to control certain genes, called oncogenes. Therefore, “multi-step” theory predicts that two or more activated oncogenes may often produce synergistic effects and cause cancer. In mid-October 1985, a study on the role of Dominique Stehelin at the Pasteur Institute was reported in the Symposium on Cooperative Genes in Paris, which showed the above-mentioned interactions between two oncogenes. Synergy between oncogenes occurred in 1983. For the first time in the year, it was only when the ras and myc oncogenes acted together to convert into fibroblasts (Nature 304 volume,