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该药可增加周围和中枢血管床受损微循环的灌注,大剂量时非特异性地抑制血管平滑肌上的α-肾上腺素受体,导致周围血管扩张,为其重要药理作用之一。在各种对象(包括人)的试验中,还观察到该药具有其他药理及血流动力学效应。如抑制血小板聚集、改善红细胞变形性和血粘度;微弱的非特异性钙离子拮抗作用及降低氧耗的活性。但它们在总效应(增进缺血组织的营养血流)中的地位尚须确定。
The drug can increase peripheral and central vascular bed impaired microcirculation perfusion, large doses of non-specific inhibition of vascular smooth muscle α-adrenoceptor, leading to peripheral vasodilation, one of its important pharmacological effects. It has also been observed in other subjects (including humans) to have other pharmacological and hemodynamic effects. Such as inhibiting platelet aggregation, improving erythrocyte deformability and blood viscosity; faint non-specific calcium antagonism and reduce oxygen consumption activity. However, their status in the overall effect of promoting nutritional blood flow in ischemic tissues has yet to be determined.