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目的 :研究桑黄抗肝纤维化作用及其机理。方法 :用CCl4 诱导大鼠肝纤维化模型 ,检测正常组、模型组和桑黄治疗组大鼠血清酶学、胶原成分及肝组织丙二醛 (MDA)含量和超氧化物歧化酶 (SOD)活性。结果 :桑黄能显著降低肝纤维化大鼠血清氨基转移酶水平和胶原含量 ,并可提高SOD活性 ,显著减少血清中的活性氧自由基。结论 :桑黄可诱导提高SOD活性 ,清除氧自由基 ,发挥抗脂质过氧化作用 ,是其抗肝纤维化作用机理之一。
Objective: To study the anti-fibrotic effect and mechanism of Sanghuang. METHODS: Rat hepatic fibrosis model was induced with CCl4. The serum enzymology, collagen composition, malondialdehyde (MDA) content and superoxide dismutase (SOD) in liver tissue were detected in normal group, model group and mulberry treatment group. active. Results: Sanghuang could significantly reduce the level of serum aminotransferase and collagen in liver fibrosis rats, increase the activity of SOD, and significantly reduce the reactive oxygen species in serum. Conclusion: Phellinus igniarius can induce SOD activity, scavenge oxygen free radicals, and exert anti-lipid peroxidation, which is one of the mechanisms of anti-hepatic fibrosis.