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重症急性胰腺炎(SAP)是以胰腺自身消化,胰腺实质坏死为病理基础的一种全身性疾病,其严重性表现为起病急、病情重、发展快、并发症多、死亡率高。近年来,其病因学及病理生理学方面研究取得了很大进展,提出了许多相关假说,发现了许多与其发生发展相关的蛋白,包括胰腺炎相关蛋白(PAP)。它在急性胰腺炎时大量合成、分泌,而在正常胰腺及胰液中并不存在,并随胰腺炎的加重和缓解而发生相应变化,故可用于指导胰腺炎的病情转归。1984年Keim等发现胰腺炎相关蛋白后,许多学者对其与急性胰腺炎的发病机制、诊断和预后等的关系进行了研究,取得一些新进展。
Severe acute pancreatitis (SAP) is a systemic disease based on the pathological basis of the pancreatic autodigestion and pancreatic parenchymal necrosis. Its severity is acute onset, severe illness, rapid development, multiple complications and high mortality. In recent years, great progress has been made in its etiology and pathophysiology. Many related hypotheses have been put forward and many proteins related to its development have been found, including pancreatitis-related protein (PAP). It is a large number of acute pancreatitis synthesis, secretion, and in the normal pancreas and pancreatic juice does not exist, and with the aggravation of pancreatitis and the corresponding changes occur, it can be used to guide the prognosis of pancreatitis. After the discovery of pancreatitis-related proteins by Keim et al. In 1984, many scholars have made some researches on its relationship with the pathogenesis, diagnosis and prognosis of acute pancreatitis. Some new progress has been made.