作者列举的中耳炎颅内并发症中包括后窝综合症(posterior fossa syndrome)。指出此类疾患发病率虽已明显下降但死亡率仍高:迷路炎为1.9%(Oeken,1960);脑膜炎6%(Kornmesser,1980);脑脓肿30%(Ganz,1980)。各种颅外并发症中面瘫、耳聋和平衡障碍可引起病人部分残废。作者指出,近10年来大部分颅内并发症是由急性中耳炎乳突炎引起,一种例外原因为具有后窝占位性症状体征的隐匿性胆脂瘤。面瘫:500例中有15例。胆脂瘤的炎性基质只有浸润神经鞘引起神经炎和运动神经元变性时才损害面神经功能。除非神经充分减压并完全根除面神经管和面神经干上的胆脂瘤基质,否则很少完全恢复功能。面神经周围气房的残余胆脂瘤可从下鼓室底向颈静脉球、耳蜗基底转和颈动脉 The authors list otitis media intracranial complications including posterior fossa syndrome. Pointed out that although the incidence of such diseases has dropped significantly but mortality is still high: labyrinthitis is 1.9% (Oeken, 1960); meningitis is 6% (Kornmesser, 1980); brain abscess is 30% (Ganz, 1980). A variety of extracranial complications in facial paralysis, deafness and balance disorders can cause some patients with disabilities. The authors note that the majority of intracranial complications in the past decade have been caused by acute otitis media mastoiditis, an exception to which is occult cholesteatoma with posterior fossa-dominant symptoms. Facial paralysis: 15 of 500 cases. The inflammatory matrix of cholesteatoma impairs facial nerve function only when infiltrating the nerve sheath causes neuritis and motor neuron degeneration. Unless the nerve decompresses completely and completely eradicates the cholesteatoma matrix on the facial and facial nerve trunks, it rarely regains its function. The residual cholesteatoma around the facial nerve may flow from the bottom of the tympanic cavity to the jugular bulb, the cochlear basal turn, and the carotid artery