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为了解阵发性室上性心动过速的产生机制，用心内电生理学方法检查３１例阵发性室上性心动过速（简称室上速）患儿。结果：房室结折返９例，均为慢快型；房室折返共２２例，其中隐匿性附加束１１例，预激综合征１０例，Ｍａｈａｉｍ束１例。异丙肾上腺素和阿托品可使双通道显示，三通道变为双通道并利于室上速的产生。如双通道和附加束同时存在，室上速开始时折返环可以不固定。提示，室上速以房室折返为主，同一病例中可以有不同的折返回路及诱发因素。
To understand the mechanism of paroxysmal supraventricular tachycardia, 31 patients with paroxysmal supraventricular tachycardia (SVS) were examined by electrophysiological method. Results: Atrioventricular nodal reentry was found in 9 cases, all of which were slow type. There were 22 cases of atrioventricular retroversion, including 11 cases of occult attachment, 10 cases of pre-excitation syndrome and 1 case of Mahaim bundle. Isoproterenol and atropine can make dual-channel display, three-channel into dual-channel and conducive to the production of supraventricular tachycardia. Such as dual-channel and additional beam coexist, supramaximal reentrant ring may not be fixed at the beginning. Prompt, supraventricular tachycardia to atrioventricular reentry-based, in the same case can have different reentrant circuits and predisposing factors.