假肥大型进行性肌营养不良症(Du-chenne Muscular Dystrophy,DMD)为一严重消耗性疾病,表现为进行性加重的肌肉萎缩和运动困难。本病的发病机制尚不十分清楚,曾提出神经源学说,血管源学说,但均因根据不足而放弃,近年的研究集中在细胞膜上。一些研究发现本病时肌细胞膜的形态、生化、生物物理特性改变,同时这种改变也存在于红细胞、皮肤成纤维细胞、淋巴细胞,因而认为是遗传因素引起的全身 Du-chenne Muscular Dystrophy (DMD) is a severe and consumptive disease characterized by progressive aggravation of muscular dystrophy and locomotor dysfunction. The pathogenesis of this disease is not yet very clear, had put forward the neurogenic theory, vascular theory, but all give up due to lack of research in recent years focused on the cell membrane. Some studies found that the muscle cell membrane morphological, biochemical, biophysical properties change, while this change also exists in red blood cells, skin fibroblasts, lymphocytes, which is considered genetic factors caused by the whole body