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中心性浆液性视网膜病的特征,是眼后极部神经感觉部视网膜下有浆液积聚。本病病因尚不明,对发病机理曾提出许多种设想,大多理由不够充足。本文从阐述色素上皮生理学的有关事实出发,提出了本病发病机理的新假说。 1.完整的视网膜色素上皮在神经感觉部视网膜与脉络膜之间构成一道屏障。视网膜毛细血管没有孔,是紧闭的。血-视网膜屏障位于视网膜血管壁内,有闭锁小带把细胞间隙封住。但脉络膜毛细管却有孔,是有漏洞的。对脉络膜血管来说,色素上皮起着血-视网膜屏障的作用。色素上皮屏障的形态基质就是把色素上皮相邻细胞间隙密封的位于顶端的闭锁小带区。这可证明如下: (1)把示踪底物如过氧化物酶注入玻璃体腔,可见其迅速弥散,移行于感光器之间,经视
The characteristic of central serous retinopathy is the accumulation of serous fluid under the retina of the posterior facial nerve sensory area. The etiology of the disease is not yet known, and many of the assumptions made about the pathogenesis have been made, mostly for inadequate reasons. Based on the related facts of pigment epithelium physiology, this article proposed a new hypothesis of the pathogenesis of this disease. 1. The intact retinal pigment epithelium forms a barrier between the retina and the choroid in the neurosensory part. Retinal capillaries without holes, is closed. Blood - retinal barrier located in the retinal blood vessel wall, a small barrier to the cell gap sealed. However, there are holes in the chorioretinal capillary, there are loopholes. For choroidal vessels, the pigment epithelium functions as a blood-retinal barrier. The morphological matrix of the pigmented epithelial barrier is the topmost, locked, small band that seals the gap between the adjacent epithelia of the pigment epithelium. This can be demonstrated as follows: (1) The tracer substrate such as peroxidase into the vitreous cavity, we can see its rapid dispersion, migration between the photoreceptors,