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血栓可分两类:一是造成静脉栓塞的“红血栓”,另一是造成动脉栓塞的“白血栓”。前者系纤维蛋白组成的网将血细胞等有形成分包埋其中,后者系由于动脉内膜损伤后使循环血液中的血小板与内膜之下的胶元接触,从而产生续发的生化反应,形成血小板的粘附与聚集。近期的报导认为,血小板的聚集是由于花生四烯酸的环状氧化物,促凝血素(TxA_2)和前列环素(PGI_2)抑制或兴奋腺甙酸环化酶,以调节血小板内cAMP水平的结果。
Thrombosis can be divided into two categories: one is the “red thrombus” that causes venous embolism, and the other is a “white thrombus” that causes arterial embolism. The former is a network composed of fibrin, which encloses blood cells and other components. The latter is caused by the contact of the platelets in the circulating blood with the glue cells under the endometrium due to the intimal injury of the arteries. Formation of platelet adhesion and aggregation. Recent reports suggest that platelet aggregation is due to arachidonic acid cyclization, pro-thrombin (TxA 2) and prostacyclin (PGI 2) inhibit or excite adenylate cyclase to regulate cAMP levels in platelets. result.