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过敏性哮喘是肺对外来致敏蛋白敏感性改变的结果。首次接触致敏蛋白后,体内即有IgE抗体形成,它吸附在细胞上,并使细胞致敏。再次接触时,该蛋白和吸附有抗体的敏感细胞起作用,促使能致支气管痉挛的介质释出。这些使支气管痉挛的物质主要是组织胺和SRS-A(慢性反应性物质-A)。现已证明它们来自肥大细胞内的颗粒。人类肥大细胞内的颗粒用电镜检查时,可分为二类:一类呈漩涡状结构,另一类呈平行线条结构。
Allergic asthma is a result of the lung’s sensitivity to exogenously sensitized proteins. After the first exposure to allergen, IgE antibodies are formed in the body, which adsorb on the cells and sensitize the cells. Upon contact again, the protein acts with sensitive, antibody-adsorbed cells to release the mediators of bronchospasm. These bronchospasm-causing substances are mainly histamine and SRS-A (Chronic Reactive Substance-A). It has been demonstrated that they come from particles within mast cells. Human mast cells within the electron microscopic examination, can be divided into two categories: one was swirling structure, the other was parallel to the line structure.