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恶性青光眼发病基础为睫状体晶体阻滞,无论曾否接受抗青光眼手术,均可发生。作者观察18例(女15,男3)20眼。抗青光眼手术后确诊的有11眼,手术前确诊的有9眼。临床表现典型,急性发作时前房中央部显著变平。眼压特续升高,房角全部闭锁。缩瞳剂不仅无效,反而有害。有2例因使用缩瞳剂诱发恶性青光眼。药物治疗仅6眼有效(30％),这可能是由于确诊较晚,不适当地长期应用缩瞳剂,房角粘连之故。其余14眼共进行28次不同手术,包括玻璃体吸出,晶体冷冻摘出术等,11眼眼压降低。视力改善者仅6眼,不变者5眼,恶化者9眼,其中6眼视力丧失。若在抗青光眼手术之前
The basis for the pathogenesis of malignant glaucoma ciliary body crystal block, whether or not anti-glaucoma surgery, can occur. The authors observed 18 (female 15, male 3) 20 eyes. There were 11 eyes diagnosed after anti-glaucoma surgery and 9 eyes were diagnosed before surgery. Typical clinical manifestations, acute central flat anterior chamber significantly flattened. Continued increased intraocular pressure, angle all locked. Miotic agent not only invalid, but harmful. 2 cases of malignant glaucoma due to the use of miotic agents. Drug treatment was effective in only 6 eyes (30%), which may be due to a later diagnosis, inappropriate long-term use of miotic agents, angle adhesions. The remaining 14 eyes underwent 28 different operations, including vitreous aspiration, crystal cryoablation and so on, 11 eyes reduced intraocular pressure. Only 6 eyes improved, 5 eyes unchanged, 9 eyes worsted, and 6 eyes lost vision. If anti-glaucoma surgery before