Atherosclerosis is an inflammatory disease characterized by leukocyte infiltration,smooth muscle cell accumulation and neointima formation[1]. Activation and damage of the endothelial monolayer seems to trigger the development of the lesions. Initially,it was thought that the damaged endothelial cells were replaced by the adjacent intact endothelium. Atherosclerosis is an inflammatory disease characterized by leukocyte infiltration, smooth muscle cell accumulation and neointima formation [1]. Activation and damage of the endothelial monolayer seems to trigger the development of the lesions. Initially, it was thought that the damaged endothelial cells were replaced by the adjacent intact endothelium.